| 赵梅莘,毛远,张卫方,张燕燕.18F-FDG PET/CT显像脾脏弥漫代谢增高原因分析[J].中国医学影像技术,2015,31(11):1716~1719 |
| 18F-FDG PET/CT显像脾脏弥漫代谢增高原因分析 |
| Reason analysis of diffuse increased splenic FDG uptake on 18F-FDG PET/CT |
| 投稿时间：2015-02-10 修订日期：2015-09-17 |
| DOI：10.13929/j.1003-3289.2015.11.029 |
| 中文关键词: 体层摄影术,发射型计算机,单光子 18F氟脱氧葡萄糖 脾脏 代谢增高 |
| 英文关键词:Tomography, emission-computed, single-photon Fluorodeoxyglucose F18 Spleen Increased uptake |
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| 摘要点击次数: 2453 |
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| 中文摘要: |
| 目的 探讨18F-FDG PET/CT显像中脾脏摄取弥漫性增高的原因。方法 以脾脏弥漫性摄取高于肝脏为研究纳入标准,脾脏SUVmax/肝脏SUVmax>1为定量指标,共纳入36例患者,结合手术病理、出院诊断及随访结果,分析患者疾病构成情况。结果 36例脾脏摄取弥漫性增高的患者中,15例淋巴瘤、3例结核、9例其他感染性疾病、1例大动脉炎、1例系统性红斑狼疮,1例自身免疫性溶血性贫血,2例特发性血小板减少性紫癜,1例结节病、3例实体肿瘤(其中2例正在接受粒细胞集落刺激因子治疗)。结论 PET/CT显像中脾脏弥漫性代谢增高的现象,在淋巴瘤、结核及其他炎症状态、贫血、结节病、正在接受粒细胞集落刺激因子的患者中均可以出现,并不是某一类疾病的特征性表现。 |
| 英文摘要: |
| Objective To explore the reason for intense splenic uptake in patients with diffuse increased splenic FDG uptake on 18F-FDG PET/CT. Methods A retrospective review identified patients with diffuse increased splenic FDG uptake greater than hepatic activity. The SUVmax for the liver and spleen were measured and then the spleen/liver ratio (S/L ratio) was calculated. Finally 36 patients with S/L ratio above 1 were included into analysis. The final diagnosis was established based on pathological biopsy, discharge diagnosis and long term follow up. The disease composition was analyzed. Results According to final diagnosis, 36 patients with diffuse increased splenic FDG uptake, included 15 with lymphoma, 3 with tuberculosis, 9 with other infectious diseases, 1 with takayasu arteritis, 1 with systemic lupus erythematosus, 1 with autoimmune hemolytic anemia, 2 with idiopathic thrombocytopenic purpura, 1 with sarcoidosis and 3 with solid tumors. In the patients with solid tumors, 2 were receiving treatment of granulocyte colony-stimulating facter (G-CSF). Conclusion Diffused increased splenic uptake on 18F-FDG PET/CT may appear in patients with lymphoma, tuberculosis or other inflammatory diseases, anemia, sarcoidosis and patient who is receiving treatment of G-CSF. It is not a characteristic feature of any specific disease. |
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